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The Expression of TRIM6 Activates the mTORC1 Pathway by Regulating the Ubiquitination of TSC1-TSC2 to Promote Renal Fibrosis

Front Cell Dev Biol. 2021-02; 
Weiwei Liu, Yang Yi, Chuanfu Zhang, Baojuan Zhou, Lin Liao, Wenrui Liu, Jing Hu, Qiming Xu, Jie Chen, Jianrao Lu
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Catalog Antibody … Inc., bs-9165R) and mouse anti-TSC1 (Santa Cruz, sc-514817) or mouse anti-TSC2 (Novus Biologicals, Inc.; Littleton, CO?… targeting rat TRIM6 and negative control (Supplementary Table 5) were designed, synthesized, annealed, and subcloned into pShuttle-H1 (GenScript)?… Get A Quote

摘要

Renal fibrosis is considered as the final pathway of all types of kidney diseases, which can lead to the progressive loss of kidney functions and eventually renal failure. The mechanisms behind are diversified, in which the mammalian target of rapamycin (mTOR) pathway is one of the most important regulatory pathways that accounts for the disease. Several processes that are regulated by the mTOR pathway, such as autophagy, epithelial-mesenchymal transition (EMT), and endoplasmic reticulum (ER) stress, are tightly associated with renal fibrosis. In this study, we have reported that the expression of tripartite motif-containing (TRIM) protein 6, a member of TRIM family protein, was highly expressed in renal fibros... More

關鍵詞

TRIM6, TSC1, TSC2, angiotensin II, mTOR, renal fibrosis
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